tci手机p331m rom如何root

来源：蜘蛛抓取(WebSpider) 时间：2014-09-06 00:04 标签： tcl p331m怎么样
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TCL A988壁纸下载
TCL A988产品报价tclp331m可以root吗_百度知道
tclp331m可以root吗
提问者采纳
您好 !TCL P331m Android os4.4 , 不是不能Root.只是暂时未有 &一键ROOT& 工具能 Root成功它.Root大师, 卓大师, 什麼什麼精灵都不行的.如果极想Root它,建议您先刷第3方Recovery, 再用刷Root.您亦可使用 Busybox-Nonroot , 在不Root 的情况下,把不喜欢的自带应用, 禁止启动 !.^^
因Root 机增加了手机数据被盗风险 ,
故不建议您使用Root 过的手机来操作网上银行或快捷支付等服务喔 !!..希望这能帮到您 !!
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目前只要是安卓系统都是可以ROOT，其中部分ROM屏蔽了ROOT,所以不能直接ROOT,但是可以直接刷机，比如MI2,而中兴手机也有不用ROOT的，，，他的权限是自动打开的，，，所有很方便，但是不是所有机型都是这样。。。你可以手机连接电脑，安装甜椒，自动获取驱动连接后，选择ROOT破解，即可，，，重启后，如果已经ROOT，直接安装SUPERUSER即可。。如果仍然不能ROOT，可以试试其他刷机工具，XIAZAIBA有很多，。，，
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出门在外也不愁cGMP and cGMP-dependent protein kinase I pathway in dorsal root gan...
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2014 Sep 1;39(19):1533-41. doi: 10.1097/BRS.0456.cGMP and cGMP-dependent protein kinase I pathway in dorsal root ganglia contributes to bone cancer pain in rats.1, , , , .1Departments of *Anesthesiology and +Pain Medicine, Affiliated Hospital of Xuzhou Medical College, Xuzhou, Jiangsu, C and ?Jiangsu Province Key Laboratory of Anesthesiology, Xuzhou Medical College, Xuzhou, Jiangsu, China.AbstractSTUDY DESIGN: A prospective, randomized experimental research.OBJECTIVE: To demonstrate the role of cGMP (cyclic guanosine monophosphate)-cGKI (cGMP-dependent protein kinase I) pathway in dorsal root ganglia (DRG) in bone cancer pain.SUMMARY OF BACKGROUND DATA: Treating bone cancer pain continues to possess a major clinical challenge because the specific cellular and molecular mechanisms underlying bone cancer pain remain elusive. cGMP and cGMP-dependent protein kinases pathway in DRG plays important role in nerve injury-induced hyperexcitability of DRG neurons, as well as neuropathic pain, however, whether this pathway participates in bone cancer pain is unknown.METHODS: The rat model of bone cancer pain was produced by intramedullary injection of rat breast cancer cells (Walker 256) into right tibia. Thermal hyperalgesia and mechanical allodynia were measured before and after administration of inhibitor of cGMP-cGKs pathway (Rp-8-pCPT-cGMPS). Immunofluorescence and reverse transcription-polymerase chain reaction were used to reflect expression of cGKI in DRG neurons, whereas the concentration of cGMP in DRG was tested using enzyme-linked immunosorbent assay method. Whole-cell patch clamp was used to record the hyperexcitability of small neurons in DRG with or without cGKs inhibitor after tumor cell implantation (TCI).RESULTS: TCI treatment significantly increased the concentration of cGMP in DRG and activity of cGKs in DRG and the spinal cord. TCI treatment also induced upregulation of cGKI messenger ribonucleic acid and protein in DRG, as well as enhanced hyperexcitability in DRG neurons. Spinal administration of Rp-8-pCPT-cGMPS, cGMP-cGKs inhibitor, significantly suppressed TCI-induced activation of cGMP-cGKI signaling, and hyperexcitability of DRG neurons. Meanwhile, in vivo intrathecal delivery of the Rp-8-pCPT-cGMPS significantly prevented and suppressed TCI-induced hyperalgesia and allodynia.CONCLUSION: From these results, we confirm that TCI treatment activates cGMP-cGKI signaling pathway and continuing activation of this pathway in DRG is required for hyperalgesia and/or hyperalgesia and allodynia after TCI treatment.LEVEL OF EVIDENCE: N/A.PMID:
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